Musculoskeletal: The Lower Extremity and Gait
Most of what a physical therapist does for the lower extremity comes down to one question: can this patient bear weight and walk safely, and if not, why not? The NPTE tests that question through hip and knee arthroplasty precautions, ligament and meniscus injuries, ankle sprains, the gait cycle and its deviations, and weight bearing orders after fracture. This chapter ties the tissue, the protocol and the gait pattern together so you can reason from findings to the safest correct answer.
By the end of this chapter you can
- Recognize hip osteoarthritis, femoroacetabular impingement, labral tears and hip fractures, and apply the total hip precautions that match the surgical approach.
- Choose and interpret the special tests for ACL, PCL, collateral ligament and meniscus injuries.
- Manage patellofemoral pain and progress rehabilitation after ACL reconstruction and total knee arthroplasty using time and criteria.
- Apply the Ottawa ankle and foot rules and distinguish lateral, medial and high ankle sprains, Achilles disorders and plantar heel pain.
- Describe foot posture and the windlass mechanism and relate pronation and supination to the gait cycle.
- Name the phases of the gait cycle, the normal joint angles and muscle activity in each, and the normal spatiotemporal values.
- Identify common gait deviations, including Trendelenburg, steppage, vaulting and circumduction, and their causes.
- Apply fracture healing timelines and the five weight bearing orders, and adapt care for osteoporosis, rheumatoid arthritis and lower limb amputation.
Section 1The hip
Hip osteoarthritis
The hip is a deep ball and socket joint whose stability comes mainly from bone, the labrum and strong capsular ligaments (iliofemoral, pubofemoral and ischiofemoral, all tightening in extension). Osteoarthritis of the hip is a disease of the whole joint: cartilage loss, subchondral sclerosis, osteophytes and capsular thickening. The classic presentation is groin or anterior thigh pain (sometimes referred to the knee), morning stiffness lasting less than an hour, pain with weight bearing and a capsular pattern of loss: internal rotation and flexion most, then abduction, with extension also limited. The hip clinical practice guideline lists features that support the diagnosis in adults over 50: moderate anterior or lateral hip pain with weight bearing, morning stiffness under an hour, and hip internal rotation and flexion each limited by more than 15 degrees compared with the other side.
Evidence based treatment includes patient education and weight management, manual therapy (mobilization and manipulation) for short term pain relief and motion, flexibility and strengthening (especially hip abductors, extensors and quadriceps), functional and balance training and an assistive device when needed. A cane is held in the hand opposite the painful hip: it moves with the involved leg and reduces the abductor force needed on the stance side, lowering joint reaction force. Carrying a load in the hand on the same side as the painful hip also reduces abductor demand, whereas carrying it on the opposite side increases it.
Femoroacetabular impingement and labral tears
Femoroacetabular impingement (FAI) is abnormal contact between the femoral head neck junction and the acetabular rim. A cam lesion is an aspherical bump at the femoral head neck junction, more common in young athletic men; it is jammed into the acetabulum in flexion and internal rotation, shearing the cartilage and labrum. A pincer lesion is acetabular overcoverage (retroversion or a deep socket), more common in middle aged women, where the rim crushes the labrum. Mixed forms are common. Patients report groin pain with prolonged sitting, deep squatting, pivoting and getting in and out of a car. The FADIR test (flexion, adduction, internal rotation) is very sensitive, so a negative test helps rule out intra articular pathology, but it is not specific.
A labral tear causes groin pain with mechanical symptoms (clicking, catching, locking), often with a C sign (the patient cups the lateral hip with thumb and fingers). Most occur in the anterosuperior labrum. Initial treatment is activity modification, avoiding end range flexion and rotation under load, core and hip strengthening and movement retraining. After arthroscopic labral repair or osteoplasty, typical precautions are protected weight bearing (often flat foot or partial weight bearing for 2 to 4 weeks), limits on flexion (often to about 90 degrees) and on external rotation and extension early (to protect the capsule), and avoidance of active straight leg raising and loaded hip flexion, which irritate the iliopsoas.
Lateral hip pain and other conditions
Greater trochanteric pain syndrome (gluteal tendinopathy, with or without trochanteric bursitis) causes lateral hip pain worse lying on the side, climbing stairs and standing on one leg. Single leg stance held for 30 seconds reproducing lateral hip pain is a useful test. Avoid compressive positions (crossing legs, hip adduction stretches such as the iliotibial band stretch) and progress isometric and then isotonic abductor loading. Remember hip conditions in children and adolescents: Legg Calve Perthes disease (avascular necrosis of the femoral head, boys about 4 to 8 years, painless limp), slipped capital femoral epiphysis (obese adolescents, often boys about 10 to 16, knee or groin pain, obligatory external rotation with hip flexion; a non weight bearing emergency referral) and developmental dysplasia (Ortolani and Barlow tests in infants).
Hip fractures
Hip fractures in older adults usually follow a fall from standing height in a person with osteoporosis. The leg is classically shortened and externally rotated. Location determines treatment because of the blood supply to the femoral head, which comes mainly from the medial circumflex femoral artery through retinacular vessels along the neck:
- Femoral neck (intracapsular) fractures can disrupt this supply, leading to avascular necrosis and nonunion. Displaced neck fractures in older adults are usually treated with hemiarthroplasty or total hip arthroplasty; nondisplaced ones with screws.
- Intertrochanteric and subtrochanteric (extracapsular) fractures have good blood supply and are fixed with a sliding hip screw or intramedullary nail.
Modern fixation and arthroplasty generally allow weight bearing as tolerated immediately, and early mobilization (within 24 to 48 hours) reduces complications such as pneumonia, pressure injury, deep vein thrombosis and delirium. Mortality within a year is high, and many patients do not return to prior function, so balance training, strengthening and fall prevention are core goals. If a hemiarthroplasty was done through a posterior approach, posterior hip precautions apply.
Total hip arthroplasty and precautions by approach
The precautions after total hip arthroplasty (THA) follow the capsule and muscles that were cut, because those positions are the ones that let the femoral head lever out of the socket. Many surgeons now relax or drop precautions, especially with the anterior approach, so the surgeon's orders always come first; on the exam, apply the classic set for the stated approach.
| Approach | Tissue disrupted | Dislocation direction | Classic precautions (often about 6 to 12 weeks) |
|---|---|---|---|
| Posterior (posterolateral) | Short external rotators, posterior capsule | Posterior | No hip flexion past 90 degrees, no adduction past midline (no crossing legs), no internal rotation; raised toilet seat, pillow between knees, avoid low chairs |
| Anterior (direct anterior) | Anterior capsule, intermuscular interval (tensor fasciae latae and sartorius) | Anterior | No hip extension past neutral, no external rotation (especially combined with extension, as in stepping back and turning outward), no bridging or prone lying with extension early per surgeon; lateral femoral cutaneous nerve numbness is common |
| Anterolateral (lateral) | Partial detachment of gluteus medius and minimus | Anterior (less common) | Avoid extension and external rotation; avoid active or resisted abduction early to protect the repaired abductors; Trendelenburg gait may persist |
Early rehabilitation after THA: ankle pumps, quadriceps and gluteal sets, heel slides within precautions, bed mobility and transfers using adaptive equipment (reacher, sock aid, long handled shoe horn), gait training with a walker progressing to a cane, and stair training (up with the good, down with the bad: the uninvolved leg leads going up, the involved leg and assistive device lead going down). Most cemented and modern cementless implants allow weight bearing as tolerated. Watch for DVT (calf pain, swelling, warmth; the Wells criteria guide referral), dislocation (sudden pain, a shortened and rotated limb, inability to bear weight), infection and leg length discrepancy.
Reasoning: A low toilet requires flexing the hip beyond 90 degrees, and reaching to the foot adds flexion and often internal rotation, the exact posterior dislocation positions. The best answer is a raised toilet seat (or commode over the toilet), backing up until she feels it, sliding the operated leg forward as she sits, and a sock aid and reacher for dressing. Teaching her to cross the operated leg over the other knee to reach her foot or to bend forward fully is wrong. Safety point: sudden severe pain with a shortened, internally rotated leg after a posterior approach suggests dislocation; stop and notify the surgeon.
Hip problems separate by location and age: groin pain with loss of internal rotation in an older adult is osteoarthritis, groin pain with sitting and pivoting in a young athlete suggests FAI or a labral tear, and lateral pain with side lying suggests gluteal tendinopathy. Fracture location decides blood supply and surgery, and precautions after arthroplasty mirror the approach: posterior means no flexion past 90, adduction or internal rotation; anterior means no extension with external rotation.
Section 2The knee
Ligament injuries
The anterior cruciate ligament (ACL) resists anterior translation of the tibia on the femur and rotational loads. Most tears are noncontact: deceleration, cutting or landing with the knee near extension, valgus collapse and tibial rotation. Patients report a pop, rapid swelling (hemarthrosis within hours) and giving way. Women have a higher risk, linked to landing mechanics, neuromuscular control and other factors; injury prevention programs that train landing, cutting and hamstring strength reduce ACL injuries. The unhappy triad classically combines ACL, MCL and medial meniscus tears, although lateral meniscus tears are actually more common with acute ACL injury.
The posterior cruciate ligament (PCL) resists posterior tibial translation; it is torn by a fall on a flexed knee with the foot plantarflexed or a dashboard injury. Isolated PCL tears are usually managed nonoperatively with quadriceps emphasis, since the quadriceps pulls the tibia anteriorly; avoid isolated resisted hamstring work early because it pulls the tibia posteriorly. The medial collateral ligament (MCL) resists valgus stress and is injured by a blow to the lateral knee; most isolated MCL tears heal well with a hinged brace and early motion. The lateral collateral ligament resists varus and is often injured with the posterolateral corner, which may need surgery.
| Test | How it is done | Positive finding | Suggests |
|---|---|---|---|
| Lachman | Knee in 20 to 30 degrees flexion, stabilize femur, translate tibia anteriorly | Increased translation, soft or absent end feel | ACL tear (most sensitive ACL test) |
| Anterior drawer | Knee at 90 degrees, foot stabilized, pull tibia forward | Increased translation | ACL tear (less sensitive acutely due to guarding) |
| Pivot shift | Valgus and internal rotation as the knee moves from extension to flexion | Clunk as the subluxed lateral tibial plateau reduces near 30 to 40 degrees | ACL tear (most specific) |
| Posterior drawer | Knee at 90 degrees, push tibia posteriorly | Increased posterior translation | PCL tear (most accurate PCL test) |
| Posterior sag (Godfrey) | Supine, hips and knees at 90 degrees | Tibia sags posteriorly | PCL tear |
| Valgus stress | Valgus force at 0 and at 30 degrees of flexion | Gapping at 30 only: MCL; gapping at 0: MCL plus cruciate or capsule | MCL injury |
| Varus stress | Varus force at 0 and 30 degrees | Lateral gapping | LCL or posterolateral corner |
| Dial test | Prone, compare external rotation at 30 and 90 degrees | Over 10 degrees more at 30 only: posterolateral corner; at both: PCL plus corner | Posterolateral corner injury |
| McMurray | Flex fully, rotate tibia and extend | Painful click along the joint line | Meniscus tear |
| Thessaly | Single leg stance in 20 degrees flexion, rotate trunk and knee | Joint line pain or catching | Meniscus tear |
| Joint line tenderness | Palpate medial and lateral joint line | Pain | Meniscus tear (sensitive, not specific) |
| Apley compression and distraction | Prone, knee at 90, compress and rotate, then distract and rotate | Pain with compression (meniscus), with distraction (ligament) | Meniscus versus ligament |
| Ober | Side lying, top hip extended, lower the leg toward the table | Leg stays abducted | Iliotibial band or TFL tightness |
Meniscus injuries
The menisci deepen the tibial plateau, spread load and assist stability. The medial meniscus is attached to the MCL and joint capsule, less mobile, and injured more often; the lateral meniscus is more mobile. Only the outer third (the red zone) has a good blood supply, so peripheral tears can be repaired while inner (white zone) tears are usually trimmed. Patients report twisting on a loaded, flexed knee, delayed swelling (over 24 hours), joint line pain and mechanical symptoms (locking, catching). A truly locked knee that cannot extend suggests a displaced bucket handle tear and needs prompt surgical referral. Degenerative tears in middle aged and older adults respond to exercise as well as to arthroscopic partial meniscectomy in most trials. After partial meniscectomy, weight bearing as tolerated and rapid progression are usual. After meniscus repair, protect the repair: limited weight bearing or a brace locked in extension for weight bearing (often about 4 to 6 weeks), limited flexion (often to 90 degrees), and no deep squatting or loaded flexion beyond 90 for about 3 to 4 months.
Patellofemoral pain
Patellofemoral pain is diffuse anterior knee pain around or behind the patella, worse with squatting, stairs (especially descending), running and prolonged sitting (the movie sign). It is common in adolescents and young adults, especially women. Contributing factors include weak hip abductors and external rotators (allowing dynamic knee valgus and femoral internal rotation under the patella), quadriceps weakness, reduced flexibility and excessive load. The Q angle (the angle between a line from the ASIS to the center of the patella and a line from the center of the patella to the tibial tubercle) averages about 13 degrees in men and 18 degrees in women; a larger angle increases lateral pull. The clinical practice guideline supports combined hip and knee strengthening as the core intervention, plus patellar taping and foot orthoses for short term relief, and education on load management. Squat and step exercises are often started in a pain free arc (open chain knee extension is gentler on the patellofemoral joint from 90 to 45 degrees and closed chain exercise from 0 to 45 degrees).
Related conditions: patellar tendinopathy (jumper's knee, pain at the inferior pole with jumping, treated with progressive isometric and heavy slow resistance or eccentric decline squats), Osgood Schlatter disease (tibial tubercle apophysitis in adolescents), iliotibial band syndrome (lateral knee pain in runners, often near 30 degrees of flexion), and patellar dislocation (usually lateral, in young women, often with a medial patellofemoral ligament tear).
ACL reconstruction rehabilitation
Common grafts are bone patellar tendon bone (strong bony fixation, more anterior knee pain and kneeling pain), hamstring tendon (less anterior knee pain, avoid aggressive hamstring loading for about 6 to 8 weeks) and quadriceps tendon. Grafts go through ligamentization: they are weakest about 6 to 12 weeks after surgery as the graft revascularizes, so although the patient feels well, the graft is vulnerable. Accelerated protocols are standard: immediate weight bearing as tolerated (if no meniscus repair), early full extension (loss of extension is the most troublesome complication; a cyclops lesion or arthrofibrosis may be the cause), quadriceps activation with neuromuscular electrical stimulation, and closed chain exercise early.
Open chain knee extension stresses the ACL graft most between about 0 and 45 degrees (the quadriceps pulls the tibia forward near extension), so it is commonly restricted early to 90 to 45 degrees; closed chain exercise in roughly 0 to 60 degrees produces low graft strain because of hamstring cocontraction and compression. Running usually begins around 3 months once there is no effusion, full motion and adequate strength. Return to sport is generally considered no earlier than about 9 months, when a battery of criteria is met: quadriceps and hamstring limb symmetry index of 90 percent or more, hop test symmetry of 90 percent or more (single, triple, crossover and timed hops), full motion, no effusion and psychological readiness. Returning before 9 months and with poor strength symmetry substantially increases reinjury risk.
Total knee arthroplasty
Total knee arthroplasty (TKA) relieves pain in advanced knee osteoarthritis. Goals after surgery: early mobilization (often the day of surgery), weight bearing as tolerated, quadriceps activation, control of swelling, and range of motion targets of full extension and at least about 90 degrees of flexion early (about 105 to 115 for stairs and rising from low chairs, about 110 for many daily tasks). Extension loss is functionally worst, so avoid a pillow under the knee; place a towel roll under the ankle instead. Quadriceps strength is markedly reduced after TKA, and early progressive strengthening plus neuromuscular electrical stimulation improves quadriceps strength and function (supported by the guideline). Continuous passive motion adds little and is not recommended routinely. Watch for DVT, infection, wound problems and arthrofibrosis; a patient who has not reached about 90 degrees by 6 to 12 weeks may be referred for manipulation under anesthesia.
Reasoning: The graft is still near its weakest point during ligamentization, and her strength is far below the 90 percent target, so cutting and pivoting are not appropriate yet. The best choice is progressive quadriceps strengthening (closed chain and open chain in a protected arc), neuromuscular and landing training, and a straight line running progression around 3 months if criteria are met. Starting sport specific cutting now or clearing her for return because motion is full is wrong. Safety point: early return and strength asymmetry strongly predict a second ACL injury, in either knee.
Knee injuries are recognized by mechanism and tests: a noncontact pivot with a pop and rapid swelling is an ACL tear (Lachman), a dashboard blow is the PCL (posterior drawer), a twist with delayed swelling and locking is the meniscus. Patellofemoral pain responds to combined hip and knee strengthening. After ACL reconstruction and TKA, restore extension and quadriceps first, and use objective criteria rather than time alone to progress.
Section 3The ankle and foot
Ankle sprains and the Ottawa rules
The lateral ankle sprain is the most common sports injury. The mechanism is inversion and plantarflexion, and the ligaments fail in sequence: the anterior talofibular ligament (ATFL, weakest and most often torn, taut in plantarflexion), then the calcaneofibular ligament (CFL), and rarely the posterior talofibular ligament. Grade I is a stretch without laxity, grade II a partial tear with some laxity, grade III a complete tear with gross laxity and marked swelling. The anterior drawer test (ATFL) and the talar tilt (inversion) test (CFL) assess laxity. Management: protection and optimal loading early, a semirigid or lace up brace for moderate to severe sprains (better than a cast), early weight bearing, range of motion, then progressive strengthening (evertors and dorsiflexors), and balance and proprioceptive training, which reduces recurrence. Up to a third or more develop chronic ankle instability.
A high ankle sprain injures the distal tibiofibular syndesmosis (anterior inferior tibiofibular ligament) by forced external rotation and dorsiflexion. It causes pain above the ankle, a positive squeeze test (compressing tibia and fibula at midcalf produces distal pain) and external rotation (Kleiger) test, and takes roughly twice as long to recover as a lateral sprain. A medial (deltoid) sprain from eversion is uncommon and often accompanies a fibular fracture.
The Ottawa ankle rules decide when x rays are needed after acute ankle trauma, with sensitivity near 100 percent for fracture. An ankle series is needed if there is pain in the malleolar zone plus any of: bone tenderness along the distal 6 cm of the posterior edge or tip of the lateral malleolus, bone tenderness along the distal 6 cm of the posterior edge or tip of the medial malleolus, or inability to bear weight both immediately and in the emergency department (four steps). A foot series is needed if there is pain in the midfoot zone plus bone tenderness at the base of the fifth metatarsal or the navicular, or inability to bear weight for four steps.
Achilles disorders
Achilles tendinopathy is either midportion (2 to 6 cm above the insertion, the area of poorest blood supply) or insertional. Risk factors include increased training load, older age, obesity, fluoroquinolone antibiotics and reduced calf strength. Morning stiffness and pain that warms up with activity are typical. The guideline supports progressive tendon loading: the eccentric heel drop program (Alfredson) or heavy slow resistance for midportion tendinopathy. For insertional tendinopathy, avoid dropping the heel below the step (dorsiflexion compresses the tendon against the calcaneus), so eccentrics are performed to floor level only. Heel lifts can reduce symptoms short term.
Achilles rupture occurs with sudden push off or landing, usually in middle aged recreational athletes ("I felt like I was kicked"). Findings: a palpable gap, weak plantarflexion (other plantarflexors still work, so active plantarflexion can be present), and a positive Thompson test (squeezing the calf produces no plantarflexion). Both operative and nonoperative care, when paired with early functional rehabilitation in a boot with heel wedges, produce similar rerupture rates. Protect from dorsiflexion past neutral for about 8 to 12 weeks and from forceful stretching for months.
Plantar heel pain
Plantar fasciitis (plantar heel pain) causes pain at the medial calcaneal tubercle that is worst with the first steps in the morning or after rest and eases with walking, then returns after prolonged standing. Risk factors include limited ankle dorsiflexion, high body mass index and prolonged standing. The windlass test (passive great toe extension in standing reproduces pain) is specific. Guideline supported interventions: manual therapy, plantar fascia specific and calf stretching, taping, prefabricated or custom foot orthoses for short term relief, night splints holding the ankle in dorsiflexion for patients with morning pain, education and strengthening. Distinguish tarsal tunnel syndrome (tibial nerve under the flexor retinaculum, burning and numbness in the sole, positive Tinel behind the medial malleolus) and a calcaneal stress fracture (positive squeeze test of the calcaneus, often in runners).
Foot fractures
| Fracture | Features | Key point |
|---|---|---|
| Ankle (bimalleolar, trimalleolar) | Rotational injuries; unstable patterns need fixation | Often non weight bearing about 6 weeks after fixation, then progressive loading |
| Fifth metatarsal avulsion (pseudo Jones) | Peroneus brevis pulls off the tuberosity during inversion | Heals well in a walking boot or hard shoe |
| Jones fracture | Metaphyseal diaphyseal junction of fifth metatarsal | Watershed blood supply, high nonunion risk; non weight bearing or fixation in athletes |
| Metatarsal stress fracture | Second metatarsal most often (march fracture), runners and military recruits | Consider relative energy deficiency and bone health in athletes |
| Navicular stress fracture | Vague dorsal midfoot pain in jumpers and sprinters | High risk; non weight bearing cast often needed |
| Calcaneal fracture | Fall from height landing on heels | Check the lumbar spine (associated compression fracture) |
| Lisfranc injury | Tarsometatarsal joint injury, plantar ecchymosis | Often missed; unstable injuries need fixation |
Foot posture and mechanics
The subtalar joint produces the triplanar motions of pronation (in open chain: eversion, abduction and dorsiflexion) and supination (inversion, adduction and plantarflexion). In closed chain the calcaneus everts while the talus adducts and plantarflexes, and the tibia rotates internally with pronation. During gait the foot pronates from initial contact through loading response to absorb shock and adapt to the ground, as the transverse tarsal joint axes become parallel and the midfoot unlocks. It then supinates from midstance through terminal stance, and the axes converge so the foot becomes a rigid lever for push off. The windlass mechanism assists: extension of the toes at heel off tightens the plantar fascia, raising the arch and inverting the calcaneus.
A pes planus (flat, overpronated) foot is flexible if the arch reappears when non weight bearing or on toe rise, and rigid if not (consider tarsal coalition). Excess pronation is associated with medial tibial stress syndrome, posterior tibial tendon dysfunction, patellofemoral pain and plantar heel pain. Posterior tibial tendon dysfunction is the commonest cause of acquired flatfoot in adults; signs include the too many toes sign from behind and inability to perform a single leg heel raise. A pes cavus (high arched, supinated) foot is rigid and poor at shock absorption, linked to lateral ankle sprains, stress fractures and neurologic disease (Charcot Marie Tooth). Other forefoot terms: hallux valgus (bunion, lateral deviation of the great toe), hallux rigidus (first MTP arthritis limiting extension, which needs about 60 degrees or more for normal push off), Morton neuroma (third web space interdigital neuroma) and claw and hammer toes.
Reasoning: He can bear weight four steps and has no Ottawa bone tenderness, so radiographs are not indicated and fracture is very unlikely. The best management is protection with a semirigid brace or lace up support, early weight bearing as tolerated, edema control and range of motion, then strengthening and balance training. Ordering films, casting or strict non weight bearing are wrong choices. Safety point: reassess if the squeeze or external rotation test is positive (syndesmosis) or if he cannot bear weight later; the rules apply to adults and need to be re applied when findings change.
Ankle and foot problems follow the mechanism and the location of pain: inversion injures the ATFL, external rotation the syndesmosis, sudden push off ruptures the Achilles. The Ottawa rules decide imaging. Tendon disorders respond to progressive loading, plantar heel pain to stretching, orthoses and night splints, and the foot changes from a mobile adapter (pronation) to a rigid lever (supination) during each step.
Section 4Normal and pathological gait
Phases and terminology
One gait cycle (a stride) runs from initial contact of one foot to the next initial contact of the same foot. It is divided into stance (about 60 percent) and swing (about 40 percent). A step is from initial contact of one foot to initial contact of the other, so one stride contains two steps. Double limb support occurs twice per cycle, each about 10 percent at normal speed (roughly 20 percent in all); as walking speed increases, double support shrinks, and running has no double support but a flight phase instead. Older adults and people with impaired balance walk with longer double support.
Two vocabularies appear on the exam. The Rancho Los Amigos terms are preferred: initial contact, loading response, midstance, terminal stance, preswing, initial swing, midswing and terminal swing. The traditional terms are heel strike, foot flat, midstance, heel off, toe off, acceleration, midswing and deceleration. They do not line up perfectly; for example traditional heel off begins terminal stance, and traditional toe off ends preswing.
Spatiotemporal values
| Measure | Typical adult value | Clinical note |
|---|---|---|
| Cadence | About 100 to 120 steps per minute (about 110) | Increases with speed; women slightly higher |
| Gait speed | About 1.2 to 1.4 m/s at comfortable pace | About 1.2 m/s commonly cited to cross a street safely; under 0.4 m/s household walker, 0.4 to 0.8 limited community, over 0.8 community walker (stroke categories); under about 1.0 m/s in older adults flags risk of falls and poor outcomes |
| Step length | About 70 cm (stride about 1.4 m) | Compare sides for asymmetry |
| Step width (base of support) | About 5 to 10 cm | Wide base in ataxia, sensory loss, fear of falling |
| Center of mass displacement | About 5 cm vertically and 5 cm side to side | Lowest at double support, highest at midstance |
| Pelvic rotation and drop | About 4 degrees forward rotation each side; about 5 degrees contralateral pelvic drop in stance | Excess drop is Trendelenburg |
Joint angles and muscle activity
Muscles in stance mostly work eccentrically to absorb and control motion; propulsion comes from the plantarflexors in terminal stance and the hip flexors in preswing and initial swing. Knowing which muscle is active in which phase lets you predict the deviation produced by its weakness.
| Phase | Hip | Knee | Ankle | Key muscle activity |
|---|---|---|---|---|
| Initial contact | About 30 degrees flexion | Near full extension (0 to 5 flexion) | Neutral | Tibialis anterior holds the ankle; hamstrings and gluteus maximus active |
| Loading response | About 30 flexion, starting to extend | Flexes to about 15 to 20 | Plantarflexes about 5 to 10 | Quadriceps eccentric (controls knee flexion), tibialis anterior eccentric (lowers forefoot), gluteus maximus and medius |
| Midstance | Extends to neutral | Extends to about 5 flexion | Dorsiflexes to about 5 to 10 | Soleus and gastrocnemius eccentric (control tibial advance), gluteus medius stabilizes pelvis |
| Terminal stance | About 10 to 20 apparent hyperextension | Near full extension | About 10 dorsiflexion, heel rises | Plantarflexors at peak; tibialis posterior and peroneals |
| Preswing | Moves toward neutral | Flexes to about 35 to 40 | Plantarflexes about 15 to 20 | Hip flexors (adductor longus, rectus femoris, iliopsoas) begin |
| Initial swing | Flexes to about 15 to 20 | Flexes to about 60 (maximum) | About 5 to 10 plantarflexion, moving up | Iliopsoas, short head of biceps femoris, tibialis anterior |
| Midswing | About 25 to 30 flexion | Extends to about 25 | Neutral | Tibialis anterior keeps toes clear |
| Terminal swing | About 30 flexion | Near full extension | Neutral | Hamstrings eccentric (decelerate the shank), quadriceps and tibialis anterior prepare for contact |
Range needed for normal walking, commonly taught: about 30 degrees hip flexion and 10 degrees apparent hyperextension, about 60 degrees knee flexion, about 10 degrees ankle dorsiflexion and 20 degrees plantarflexion, and about 60 degrees or more first MTP extension. Stairs need more knee flexion (about 90 to 105) and rising from a standard chair about 90 to 105 degrees of knee and hip flexion.
Common gait deviations and their causes
| Deviation | What you see | Usual causes |
|---|---|---|
| Trendelenburg gait | Pelvis drops on the swing side during stance on the weak side; compensated form leans the trunk over the stance limb | Weak gluteus medius on the stance side, superior gluteal nerve injury, hip OA (lean reduces joint load) |
| Gluteus maximus gait | Backward trunk lean at initial contact | Weak hip extensors (inferior gluteal nerve) |
| Quadriceps weakness | Forward trunk lean or hand pushing on thigh, knee kept extended or hyperextended in loading response | Femoral nerve palsy, L3 to L4 radiculopathy, after TKA |
| Genu recurvatum | Knee snaps back in stance | Quadriceps weakness, plantarflexion contracture, plantarflexor spasticity |
| Foot slap | Forefoot slaps after initial contact | Mild dorsiflexor weakness (eccentric control lost) |
| Steppage gait | Excess hip and knee flexion in swing to clear a dropped foot, toe first contact | Foot drop: common fibular nerve palsy, L4 to L5 radiculopathy, peripheral neuropathy |
| Circumduction | Swing limb moves in an arc to the side | Functionally long limb: knee extension contracture or brace locked in extension, plantarflexion contracture, weak hip flexors or dorsiflexors, hemiplegia |
| Hip hiking | Pelvis elevates on the swing side | Same as circumduction, or weak hamstrings so knee cannot flex |
| Vaulting | Rises on the toes of the stance limb so the other limb can clear | Long swing limb (locked knee, prosthesis too long, poor knee flexion) |
| Antalgic gait | Shortened stance on the painful limb, shorter step of the opposite limb | Pain anywhere in the limb |
| Hip flexion contracture | Forward trunk lean or increased lumbar lordosis, short step of the opposite limb, no terminal stance extension | Hip OA, prolonged sitting, transfemoral amputation positioning |
| Weak plantarflexors | No heel rise, excess dorsiflexion and knee flexion in stance, short opposite step | Tibial nerve palsy, S1 radiculopathy, Achilles rupture |
| Scissoring | Swing limb crosses the midline | Hip adductor spasticity (cerebral palsy) |
| Ataxic gait | Wide base, irregular, staggering | Cerebellar lesion or proprioceptive loss (worse with eyes closed if sensory) |
| Parkinsonian gait | Shuffling, reduced arm swing, festination, freezing | Parkinson disease |
Reasoning: Excessive hip and knee flexion in swing with toe first contact is a steppage gait from foot drop. The common fibular nerve wraps the fibular neck and is easily injured there; the deep branch supplies tibialis anterior and the toe extensors, and sensation over the dorsum of the foot and first web space fits. The best intervention is a posterior leaf spring or other ankle foot orthosis, plus education to protect the insensate skin. Circumduction training, hip flexor strengthening alone or a heel lift are wrong. Safety point: after a fibular head fracture, also check for compartment syndrome early on.
Learn the gait cycle by phase: what each joint is doing and which muscle controls it. Weakness shows up in the phase where that muscle is supposed to work (quadriceps and dorsiflexors at loading response, gluteus medius in single limb stance, plantarflexors in terminal stance, dorsiflexors in swing), and a functionally long limb in swing produces circumduction, hip hiking or vaulting.
Section 5Fractures, weight bearing and orthopedic special populations
Fracture healing
Bone heals by two routes. Secondary (indirect) healing with callus occurs when there is some motion at the fracture, as with casting, intramedullary nails or external fixation. Primary (direct) healing without visible callus occurs with rigid compression plating, where osteons cross the fracture directly. Secondary healing passes through overlapping stages.
Factors that delay healing: smoking, diabetes, poor nutrition, older age, NSAIDs in some studies, corticosteroids, infection, poor blood supply (scaphoid, femoral neck, talus, fifth metatarsal Jones zone, distal tibia), excessive motion and open fractures. A delayed union is slower than expected; a nonunion has stopped healing (often after about 6 to 9 months). Malunion heals in a poor position. Watch after any fracture for compartment syndrome (pain out of proportion and with passive stretch), fat embolism (after long bone fractures, typically 24 to 72 hours: confusion, dyspnea, petechiae), DVT and pulmonary embolism, and complex regional pain syndrome. Exercise prescription during immobilization keeps the joints above and below moving and maintains strength in the rest of the body.
Weight bearing status
| Order | Meaning | Suitable gait pattern and device |
|---|---|---|
| Non weight bearing (NWB) | No weight on the limb; foot does not touch the floor | Three point gait with walker or axillary crutches; requires good upper body strength and balance |
| Toe touch or touch down (TTWB, TDWB) | Foot touches for balance only, essentially no weight | Walker or crutches, three point pattern |
| Partial (PWB) | A stated percentage or amount, for example 50 percent of body weight | Walker or crutches, three point gait; teach with a bathroom scale |
| Weight bearing as tolerated (WBAT) | Any amount the patient can tolerate | Walker, crutches or cane as needed, wean as able |
| Full (FWB) | No restriction | Device only for balance or pain |
Weight bearing orders come from the surgeon and depend on fixation stability more than on the fracture alone. Most hip fractures and arthroplasties allow weight bearing as tolerated; intra articular fractures (tibial plateau, pilon, calcaneus), unstable ankle fractures and many osteotomies require non weight bearing or touch down weight bearing for about 6 to 12 weeks. Patients poorly judge partial weight bearing, so teach it with a scale and recheck. If a patient cannot maintain a NWB restriction safely, a wheelchair may be the safer mobility choice. For gait patterns: three point gait (device and involved limb together) for a restricted limb; two point and four point gaits (reciprocal, with each crutch moving with the opposite limb) for bilateral weakness; swing to and swing through for bilateral lower extremity paralysis. On stairs with crutches, go up with the good limb first and down with the crutches and involved limb first.
Osteoporosis
Osteoporosis is low bone mass with microarchitectural deterioration. The World Health Organization defines it by DXA T score (comparison with young adult peak bone mass): normal T score at or above negative 1.0; osteopenia (low bone mass) between negative 1.0 and negative 2.5; osteoporosis at or below negative 2.5; severe osteoporosis at or below negative 2.5 with a fragility fracture. Z scores compare with age matched peers and are used in children and premenopausal adults. Common fragility fractures: vertebral compression (often thoracic, causing kyphosis and height loss), distal radius (Colles) and hip.
Exercise for bone health combines progressive resistance training, impact or weight bearing activity as tolerated, balance training and posture work (back extensor strengthening reduces vertebral fracture risk). For people with osteoporosis, avoid loaded spinal flexion (sit ups, toe touches, deep forward bending with rotation, some yoga poses) and sudden twisting, which raise vertebral compression fracture risk. Teach hip hinge lifting, home fall prevention and adequate calcium and vitamin D. Pain with mild trauma or a sudden onset of thoracic pain in an older adult with osteoporosis suggests a compression fracture and needs medical evaluation.
Rheumatoid arthritis
Rheumatoid arthritis (RA) is a systemic autoimmune inflammatory arthritis of the synovium. Contrast it with osteoarthritis:
| Feature | Rheumatoid arthritis | Osteoarthritis |
|---|---|---|
| Pathology | Autoimmune synovitis, pannus erodes cartilage and bone | Degenerative whole joint disease, cartilage loss, osteophytes |
| Joints | Symmetric small joints: MCP, PIP, wrist, MTP; cervical spine (C1 to C2) | Asymmetric weight bearing joints, DIP, PIP, thumb base, hip, knee, spine |
| Morning stiffness | More than about an hour | Under about 30 minutes |
| Systemic signs | Fatigue, fever, weight loss, nodules, anemia, lung and eye disease | None |
| Labs | Rheumatoid factor, anti CCP, raised ESR and CRP | Normal |
| Hand deformities | Ulnar drift, swan neck, boutonniere | Heberden and Bouchard nodes |
During an acute flare (warm, swollen, painful joints), use rest, splinting in a functional position, gentle active or active assisted range of motion (one or two repetitions to maintain motion), isometrics and joint protection; avoid vigorous stretching and resisted exercise through inflamed joints. Between flares, use aerobic exercise, low load strengthening, joint protection and energy conservation. Long term corticosteroid use causes osteoporosis and skin fragility. Atlantoaxial instability from transverse ligament laxity is a serious risk: avoid cervical manipulation and end range upper cervical flexion, and report symptoms such as occipital headache, dizziness, bilateral paresthesia or gait change as possible cord compression.
Amputation level overview
Lower limb amputations in adults are mostly due to vascular disease and diabetes; trauma and tumor are the main causes in younger people. Energy cost of walking rises as the level becomes more proximal, and bilateral and transfemoral amputees often need more help.
| Level | Key points |
|---|---|
| Partial foot (toe, transmetatarsal, Chopart) | Low energy cost; Chopart tends to an equinus contracture |
| Syme (ankle disarticulation) | End bearing heel pad, limited prosthetic clearance |
| Transtibial | Energy cost roughly 20 to 40 percent above normal; avoid knee flexion contracture (no pillow under the knee, no prolonged sitting with knee bent) |
| Knee disarticulation | End bearing, long lever, knee center lower than the other side |
| Transfemoral | Energy cost roughly 60 to 100 percent above normal; avoid hip flexion, abduction and external rotation contracture (no pillow between the thighs or under the residual limb, prone lying daily) |
| Hip disarticulation and hemipelvectomy | Highest energy cost; many patients use a wheelchair |
Preprosthetic care: residual limb shaping with figure of eight elastic wrapping (tighter distally, diagonal turns, no circular turns that form a tourniquet) or a shrinker, positioning to prevent contractures, strengthening (especially hip extensors and abductors), desensitization, skin inspection of both limbs (the remaining foot is at high risk in diabetes), cardiovascular training and transfer and wheelchair training. Phantom limb sensation is nearly universal and harmless; phantom pain responds to mirror therapy, graded motor imagery and desensitization. A rigid removable dressing protects transtibial limbs and limits edema.
Reasoning: Repeated or loaded spinal flexion raises compressive force on the anterior vertebral bodies and increases the risk of vertebral compression fracture in osteoporosis. The best program combines progressive resistance training, weight bearing or impact activity as tolerated, balance training and spinal extensor strengthening, with neutral spine core work and hip hinge mechanics. Crunches, toe touches and sit ups with rotation are wrong choices. Safety point: sudden mid back pain after minor strain or a cough in this patient should be evaluated as a possible compression fracture.
Bone heals in overlapping stages, with clinical union around 6 to 8 weeks in many adult fractures and remodeling for months; weight bearing follows the surgeon's view of fixation stability, and the gait pattern and device follow the order. Osteoporosis calls for loading without loaded flexion, RA for joint protection during flares and caution in the upper cervical spine, and amputation care for contracture prevention, limb shaping and protection of the other foot.
The chapter in one breath
- Hip osteoarthritis limits internal rotation and flexion first; a cane goes in the opposite hand.
- Cam FAI affects young men, pincer FAI middle aged women; labral tears click and catch.
- Femoral neck fractures threaten the blood supply to the head; most hip fracture repairs allow weight bearing as tolerated and early mobilization.
- Posterior THA: no flexion past 90, adduction past midline or internal rotation; anterior THA: no extension or external rotation.
- Lachman is the most sensitive ACL test, pivot shift the most specific, posterior drawer the best PCL test; McMurray and Thessaly test the meniscus.
- Patellofemoral pain responds to combined hip and knee strengthening; after ACL reconstruction, restore extension first and return to sport at about 9 months with 90 percent symmetry.
- After TKA, prioritize full extension, quadriceps strength (with NMES) and at least 90 degrees of flexion.
- The Ottawa rules use malleolar, fifth metatarsal and navicular tenderness and four step weight bearing to decide on x rays.
- Achilles tendinopathy and plantar heel pain respond to progressive loading and stretching; the Thompson test detects Achilles rupture.
- Stance is about 60 percent of the gait cycle; weakness shows up in the phase where a muscle normally works (gluteus medius, Trendelenburg; dorsiflexors, steppage).
- Bone reaches clinical union around 6 to 8 weeks; osteoporosis is a T score of negative 2.5 or lower and loaded flexion is avoided.
- Transfemoral amputees risk hip flexion and abduction contractures, transtibial amputees knee flexion contractures.
Words worth knowing
| Term | What it means |
|---|---|
| Capsular pattern of the hip | Loss of internal rotation and flexion first, then abduction, from capsular restriction. |
| Femoroacetabular impingement | Abnormal contact between the femoral head neck junction and acetabular rim, as cam or pincer types. |
| FADIR test | Flexion, adduction and internal rotation of the hip to provoke intra articular pain. |
| Avascular necrosis | Bone death from loss of blood supply, a risk after femoral neck and scaphoid fractures. |
| Hemiarthroplasty | Replacement of only the femoral head, often for displaced femoral neck fractures. |
| Lachman test | Anterior tibial translation at 20 to 30 degrees flexion to test the ACL. |
| Pivot shift | A clunk of the lateral tibial plateau reducing during flexion, specific for ACL tear. |
| Posterolateral corner | LCL, popliteus and related structures that resist varus and external rotation. |
| Red zone | The vascular outer third of the meniscus, where tears can heal after repair. |
| Q angle | Angle between the quadriceps line of pull and the patellar tendon, larger in women. |
| Ligamentization | Remodeling of an ACL graft into ligament-like tissue, weakest at about 6 to 12 weeks. |
| Limb symmetry index | Involved limb performance as a percentage of the uninvolved limb. |
| Syndesmosis | Fibrous joint between distal tibia and fibula, injured in a high ankle sprain. |
| Ottawa ankle rules | Decision rule using bone tenderness and weight bearing to decide on ankle or foot x rays. |
| Thompson test | Calf squeeze that produces no plantarflexion when the Achilles is ruptured. |
| Windlass mechanism | Toe extension tightening the plantar fascia to raise the arch and stiffen the foot. |
| Jones fracture | Fracture at the metaphyseal diaphyseal junction of the fifth metatarsal with poor healing. |
| Double limb support | Periods when both feet are on the ground, about 10 percent twice per cycle. |
| Trendelenburg gait | Contralateral pelvic drop in stance from weak gluteus medius on the stance side. |
| Steppage gait | Excess hip and knee flexion in swing to clear a dropped foot. |
| Vaulting | Rising onto the toes of the stance limb to clear a long swing limb. |
| Nonunion | A fracture that has stopped healing without bony union. |
| Three point gait | Device and involved limb advance together, then the uninvolved limb. |
| T score | Bone density compared with young adult peak, in standard deviations. |
| Pannus | Inflamed synovial tissue in rheumatoid arthritis that erodes cartilage and bone. |
| Phantom limb pain | Painful sensation perceived in the amputated part. |