๐Ÿ“– Contents Chapter 13 · Assessing the Musculoskeletal and
Health Assessment · Chapter 13

Assessing the Musculoskeletal and Neurologic Systems

These two systems are assessed together because they fail together. A patient who cannot lift an arm may have a torn rotator cuff, a compressed nerve root, or an infarct in the contralateral motor cortex — and the exam that tells those apart is the same exam, performed in order, compared side to side, and repeated over time. This chapter teaches the actual hands: how to inspect a joint, grade a muscle from 0 to 5, take a Glasgow Coma Scale score that another nurse can reproduce, elicit a patellar reflex and grade it 0 to 4+, and run a neurovascular check on a casted limb in any skin tone. It also teaches the findings that end the exam and start a phone call — pain out of proportion under a cast, a new pronator drift, a widening pulse pressure with a falling heart rate — and exactly how to hand those off in SBAR.

By the end of this chapter you can

  • Perform a screening musculoskeletal exam in the correct order — inspection, palpation, range of motion, strength — and state why the abdomen is the one region where the classic four-step sequence is reordered.
  • Grade muscle strength on the 0–5 scale and deep tendon reflexes on the 0–4+ scale, and document both so another clinician gets the same number.
  • Measure and describe joint range of motion in degrees for the cervical spine, shoulder, elbow, hip, knee and ankle, and distinguish active from passive ROM.
  • Assess gait, balance and fall risk using the Timed Up and Go, the 4-stage balance test and orthostatic vital signs, using exact cutoffs.
  • Determine level of consciousness with AVPU and the Glasgow Coma Scale, and screen cognition and delirium with Mini-Cog and the CAM.
  • Test cranial nerves I–XII, motor and cerebellar function, the sensory system and reflexes, and interpret findings as upper versus lower motor neuron.
  • Describe expected variations across the lifespan — infant primitive reflexes, fontanels, pregnancy, the older adult — and assess pallor, cyanosis and erythema accurately in dark skin tones.
  • Recognize the red flags that require escalation now — compartment syndrome, cauda equina syndrome, autonomic dysreflexia, stroke by BE-FAST, rising intracranial pressure — and escalate using SBAR.
  • Apply delegation rules: what a UAP may do with a mobility-impaired or neurologically changing patient and what stays with the RN.

Section 1The Musculoskeletal Exam — Structure, Sequence and Strength

Labeled diagram of a synovial joint showing articular cartilage, joint capsule, synovial membrane, synovial cavity and adjacent bone ends
Reference. Most of what you palpate at the bedside — effusion, warmth, crepitus, tenderness — comes from the synovial joint’s capsule, membrane and cartilage. BruceBlaus, CC BY-SA 4.0 (via Wikimedia Commons)

What you are actually assessing

The musculoskeletal system is 206 bones, roughly 640 skeletal muscles, and the joints, tendons, ligaments, bursae and cartilage that connect them. At the bedside you are almost never assessing all of it. You are doing one of two exams. The screening exam is what you perform on admission and at the start of a shift on a stable patient: observe posture and gait, watch the patient move in and out of bed and reach overhead, look at the hands, and check symmetry. It takes about ninety seconds and it is enough to catch asymmetry, guarding, deformity and functional loss. The focused exam is what you perform when the screening exam, the history or the diagnosis points somewhere: a fall with hip pain, a new cast, a shoulder that will not abduct, a complaint of numb fingers at night.

Two habits make the musculoskeletal exam reliable. First, always compare side to side. The patient’s own opposite limb is the only control group you have, and it is a very good one. A knee that measures three centimeters larger in circumference than its partner is an objective finding; “knee looks swollen” is an opinion. Second, examine the joint above and the joint below the painful one. Hip pathology refers pain to the knee, cervical pathology refers pain to the shoulder and hand, and a patient who points at one place is not always pointing at the problem.

Position matters more than students expect. Expose the joint fully — you cannot assess what is under a gown or a blanket — while draping everything you are not examining. Ask about pain before you touch, and examine the non-painful side first so the patient learns what your hands feel like on tissue that does not hurt. Never force a joint through a range the patient cannot reach voluntarily; passive range of motion is performed slowly, to the point of resistance or the report of pain, and then you stop.

The sequence — and the one region that breaks it

The classic physical assessment sequence is inspection, palpation, percussion, auscultation, and it is used in that order in every body region except the abdomen. In the abdomen the order becomes inspection, auscultation, percussion, palpation, because percussing and palpating first stimulates the bowel and alters the bowel sounds you were about to listen for — you would be auscultating a gut you already provoked. Nowhere else does that problem exist, so nowhere else is the order changed.

Applied to a joint, the four steps look like this. Inspection: with the patient upright and the joint exposed, compare size, contour, alignment and symmetry with the opposite side; look for swelling, redness, muscle wasting, deformity and the way the patient holds the part at rest. Palpation: using the pads of the fingers and the back of your hand for temperature, palpate the joint line, the bony landmarks, the tendon insertions and the surrounding muscle for warmth, effusion, tenderness, nodules and crepitus — a palpable or audible grating with movement. Percussion: in the musculoskeletal and neurologic exam, percussion is done with the reflex hammer over tendons (Section 4) and, in specific situations, over the spinous processes to reproduce focal bony tenderness. Auscultation: limited but real — auscultate the carotids for a bruit before any vigorous neck range of motion in an older adult, and listen over a joint replacement or a suspected arteriovenous malformation if there is a clinical reason.

Then two steps that belong specifically to the musculoskeletal exam are added after palpation: range of motion (active first, then passive if active is limited) and muscle strength against resistance. Active before passive is not arbitrary. Active ROM tells you what the patient can do — a functional answer. If active ROM is full, you do not need passive ROM. If active ROM is limited but passive ROM is full, the joint moves and the problem is muscle, nerve, pain or effort. If both are limited, the problem is in the joint itself: contracture, effusion, mechanical block, ankylosis.

Pearl. If a patient cannot lift the arm at all but you can lift it for them through full range with no resistance, the joint is fine — look at the deltoid, the axillary nerve, the C5 root, or the motor cortex. Active-limited with passive-full is a nerve-and-muscle finding, not a joint finding.

Grading with numbers, not adjectives

Muscle strength is graded 0 to 5 against gravity and resistance, and every nurse in the building uses the same scale. Test the muscle group in isolation, stabilize the joint above it, and apply resistance through the mid-range while the patient holds. Grade each side separately: “5/5 right, 4/5 left” is information; “strength good” is not. Range of motion is recorded in degrees, from a defined zero (anatomic position) to the end of movement. You do not need a goniometer for a screening exam, but you do need to know what full looks like so you can say “knee flexion approximately 90 degrees, limited by pain” instead of “knee doesn’t bend all the way.”

GradeWhat the patient doesCommon shorthand
5Full ROM against gravity and full resistanceNormal (5/5)
4Full ROM against gravity and some resistanceGood (4/5)
3Full ROM against gravity only — no resistance toleratedFair (3/5)
2Full ROM only with gravity eliminated (limb supported sideways)Poor (2/5)
1Palpable or visible flicker of contraction, no joint movementTrace (1/5)
0No detectable contractionZero (0/5) — paralysis
Joint and motionExpected adult rangeWhat limitation may mean
Cervical flexion / extension45° / 55°Muscle spasm, arthritis; rigidity plus fever suggests meningeal irritation
Cervical rotation / lateral bending70° each side / 40° each sideDegenerative disc disease, torticollis
Shoulder flexion and abduction180°Painful arc 60–120° suggests rotator cuff impingement
Elbow flexion / extension150–160° / to 0°Effusion, contracture, heterotopic ossification
Wrist flexion / extension80–90° / 70°Carpal tunnel syndrome, rheumatoid synovitis
Hip flexion (knee flexed / knee straight)120° / 90°Osteoarthritis, effusion, flexion contracture
Knee flexion130–150°Effusion, meniscal tear, post-arthroplasty stiffness
Ankle dorsiflexion / plantar flexion20° / 45°Achilles pathology, cast immobilization, foot drop
Lumbar flexion / extension75–90° / 30°Disc disease, spasm, ankylosing spondylitis

How to chart it

Musculoskeletal documentation fails in a predictable way: it is written as a conclusion instead of an observation. “Musculoskeletal within normal limits” is unfalsifiable, unhelpful in court, and useless to the nurse who follows you and needs to know whether the left grip changed. Write what you did, what you measured, and on which side.

Vague entryPrecise entry a nurse can act on
“MSK WNL. Moves all extremities.”“Moves all four extremities to command. Strength 5/5 bilateral grips, 5/5 bilateral plantar flexion and dorsiflexion. No joint swelling, deformity or tenderness. Gait steady with single-point cane, no assist required.”
“Right knee swollen and sore.”“Right knee warm to touch compared to left, circumference 42 cm right vs 38.5 cm left at mid-patella. Positive bulge sign. Active flexion 85° limited by pain rated 6/10; passive flexion 95°. Ambulates 10 feet with walker, antalgic gait favoring right. Left knee unremarkable.”
“Weak on the left.”“Left grip 3/5, right grip 5/5. Left elbow flexion 4/5, right 5/5. Left leg 4/5 hip flexion, 5/5 elsewhere. New compared to 0800 assessment (all 5/5). Provider notified 1412.”
Interactive Build the four-step assessment sequence for any region โ€” discovering the abdomen exception the hard way โ€” then grade muscle strength 0 to 5 from what the patient actually does and read range of motion in degrees against the expected adult range. Open full ↑
Key point

Inspect, palpate, percuss, auscultate in every region except the abdomen — and in the musculoskeletal exam add active range of motion, then passive, then graded strength, always comparing left to right.

Section 2Region by Region — Spine, Limbs, Gait and the Neurovascular Check

The Adams forward bend test makes a rotational spinal deformity visible as an asymmetric rib hump that is invisible when the patient stands upright.
Reference. The Adams forward bend test makes a rotational spinal deformity visible as an asymmetric rib hump that is invisible when the patient stands upright. Rigo M, Negrini S, Weiss HR, Grivas TB, Maruyama T, Kotwicki T, CC BY 2.0 (via Wikimedia Commons)

Spine and posture

Inspect the spine with the patient standing and undressed to the underwear, from behind and from the side. From behind, the spinous processes should form a straight vertical line; the shoulders, scapulae and iliac crests should be level; the gluteal fold should be symmetric. From the side, expect the normal curves: cervical concave, thoracic convex, lumbar concave. Kyphosis is an exaggerated thoracic convexity, lordosis an exaggerated lumbar concavity, scoliosis a lateral curvature with vertebral rotation. Palpate down the spinous processes and the paravertebral muscles for tenderness and spasm.

Screen for scoliosis with the Adams forward bend test: the patient stands with feet together and bends forward at the waist with knees straight and arms hanging. You look tangentially across the back for a unilateral rib hump or lumbar prominence, which reveals the vertebral rotation that a standing exam hides. Adolescent idiopathic scoliosis is screened around ages 10 and 12 in girls and 13 or 14 in boys, because curves progress during the growth spurt. Test lumbar range of motion — flexion 75–90°, extension 30°, lateral bending 35° each way, rotation 30° each way — stabilizing the pelvis so the motion comes from the spine.

For radicular low back pain, the straight leg raise is the test to know. The patient lies supine; you raise the extended leg by the heel with the knee straight. Reproduction of the patient’s shooting pain below the knee between 30 and 70 degrees of elevation is a positive test suggesting lumbar nerve root irritation (usually L5 or S1). Hamstring tightness or posterior thigh pulling is not a positive test. Pain reproduced in the affected leg when you raise the opposite leg (crossed straight leg raise) is less sensitive but much more specific for disc herniation.

Upper extremity

At the shoulder, inspect for the squared-off contour of a dislocation and for deltoid wasting. Palpate the sternoclavicular and acromioclavicular joints, the bicipital groove and the subacromial space. Screen ROM with three commands that cover everything: “raise both arms straight overhead” (flexion and abduction to 180°), “touch the back of your neck” (external rotation and abduction), “touch the middle of your back” (internal rotation and adduction). Pain only between 60 and 120 degrees of abduction — a painful arc — suggests subacromial impingement. In the drop arm test, you passively abduct the arm to 90 degrees and ask the patient to lower it slowly; if the arm drops abruptly, suspect a rotator cuff tear.

At the elbow, palpate the olecranon and the epicondyles; tenderness over the lateral epicondyle with resisted wrist extension suggests lateral epicondylitis. At the wrist and hand, inspect for the ulnar deviation and swan-neck or boutonniere deformities of rheumatoid arthritis, the hard Heberden nodes at the distal interphalangeal joints and Bouchard nodes at the proximal interphalangeal joints of osteoarthritis, and thenar wasting. For suspected carpal tunnel syndrome, Phalen test holds the wrists in full flexion back to back for 60 seconds and is positive if numbness or tingling develops in the thumb, index, middle and radial half of the ring finger; Tinel sign taps lightly over the median nerve at the volar wrist and is positive if the same tingling shoots into those digits.

Lower extremity and gait

At the hip, inspect stance and leg length, palpate the greater trochanter for bursal tenderness, and test flexion, internal rotation (40°) and external rotation (45°) — internal rotation is usually lost first in hip osteoarthritis. The Trendelenburg sign is positive when the patient stands on one leg and the opposite hip drops, indicating weakness of the gluteus medius on the weight-bearing side. At the knee, inspect for effusion and quadriceps atrophy, palpate the joint line, and use the bulge sign for a small effusion: milk fluid upward from the medial side, then stroke down the lateral side and watch for a fluid wave to appear medially. Larger effusions are detected by ballottement of the patella. Ligament and meniscal testing — anterior drawer and Lachman for the anterior cruciate ligament, McMurray for the meniscus — is generally performed by the provider, but you should recognize the names in the chart.

Then watch the patient walk, because gait integrates everything. Observe a full walking cycle from the front, side and back for stride length, arm swing, foot clearance, base width and symmetry. Named abnormal gaits carry diagnoses: antalgic (short time in stance on the painful side), steppage (high knee lift to clear a dropped foot — peroneal nerve or L5), shuffling with reduced arm swing (parkinsonian), wide-based and unsteady (cerebellar ataxia), scissoring (spastic diplegia), and the stiff circumducted swing of a hemiparetic gait after stroke.

Fall risk deserves exact numbers. In the Timed Up and Go (TUG), the patient rises from a standard armchair, walks 10 feet (3 meters), turns, walks back and sits down, wearing usual footwear and using their usual assistive device. 12 seconds or more indicates increased fall risk (CDC STEADI). The 4-Stage Balance Test asks for feet together, semi-tandem, full tandem and single-leg stance, 10 seconds each; inability to hold full tandem for 10 seconds is a fall-risk finding. Orthostatic vital signs are positive when, within 3 minutes of standing, systolic BP falls 20 mmHg or more, diastolic BP falls 10 mmHg or more, or heart rate rises 20 beats/min or more. Measure supine after 5 minutes rest, then sitting, then standing — and stay with the patient the whole time.

Pearl. The single best predictor of a fall in the next year is a fall in the last year. Ask every adult over 65 three questions at admission: Have you fallen in the past year? Do you feel unsteady when standing or walking? Do you worry about falling? Any “yes” earns a TUG and a gait assessment.

The neurovascular check — and skin tone

Any patient with a fracture, cast, splint, traction, surgical extremity, circumferential dressing or vascular procedure gets scheduled neurovascular checks distal to the injury, compared to the opposite limb. The elements are colour, temperature, capillary refill, pulses, sensation, movement, pain and edema — often charted as CMS (circulation, motion, sensation). Expected: warm, colour matching the other limb, capillary refill under 2 seconds (longer than 2 seconds is abnormal, and 3 seconds or more is clearly delayed), palpable distal pulse equal to the other side, intact light touch and two-point sensation, active wiggle of fingers or toes, and pain that is controlled by the ordered analgesia. Frequency is typically every hour for the first 24 hours after casting or surgery, then every 4 hours, or per facility protocol — and immediately with any patient complaint.

Colour findings must be assessed correctly in every skin tone, because “pink” is a description of light skin, not a physiologic standard. In darker skin, pallor appears as ashen or grey rather than white; cyanosis appears as grey, dusky or blue-black. The reliable sites in every patient are the oral mucosa and tongue, the conjunctivae, the palms and soles, and the nail beds. Jaundice is best seen in the sclera (against the hard palate and palms if the sclera is equivocal), and in darker skin the sclera may have normal yellowish fat deposits near the periphery — look near the cornea. Erythema and early inflammation may be invisible in darker skin, so assess by palpation: compare the area to surrounding skin for warmth, induration, edema, tautness and tenderness, and use natural or full-spectrum light rather than fluorescent. Ask the patient what their normal skin colour looks like.

Neurovascular elementExpected findingAbnormal finding and what it suggests
ColourMatches the contralateral limb; oral mucosa, conjunctivae, palms/soles and nail beds unchanged from this patient’s own baseline — never judged against “pink”Newly pale, ashen or grey against the other limb → arterial compromise; dusky, grey or blue-black at mucosa, conjunctivae or nail beds → venous congestion or hypoxia
TemperatureWarm, equal to opposite limb (back of hand)Cool distal to injury → arterial insufficiency or tight cast
Capillary refillUnder 2 secondsLonger than 2 seconds, and certainly 3 or more → poor perfusion (also cold environment, shock)
PulsesPalpable, 2+ and equal bilaterallyDiminished or absent → late, ominous sign of compartment syndrome or arterial injury
SensationIntact light touch, no numbness or tinglingParesthesia, numbness → nerve compression — an early compartment sign
MovementWiggles digits actively and fullyWeak or absent movement → late nerve or muscle ischemia
PainRelieved by ordered analgesia and elevationPain out of proportion, unrelieved, worse with passive stretch of the digits → compartment syndrome until proven otherwise
Interactive Set every element of a neurovascular check on a casted limb and find out which signs are early and which are already late, then score fall risk with the Timed Up and Go, the tandem stance and orthostatic vital signs โ€” with colour findings assessed correctly in every skin tone. Open full ↑
Key point

Regional musculoskeletal assessment is comparison plus function — measure the joint in degrees, watch the patient walk, and on any immobilized limb run a scheduled neurovascular check whose colour findings you assess at the mucosa, conjunctivae, palms and nail beds rather than by expecting “pink.”

Section 3The Neurologic Exam Begins — Consciousness, Cognition and Cranial Nerves

The twelve cranial nerves emerge in a fixed order from the inferior surface of the brain, and testing them in that order keeps the exam reproducible.
Reference. The twelve cranial nerves emerge in a fixed order from the inferior surface of the brain, and testing them in that order keeps the exam reproducible. OpenStax, CC BY 4.0 (via Wikimedia Commons)

Level of consciousness first, always

Level of consciousness is the single most sensitive indicator of neurologic change, and it is assessed before anything else, because the rest of the exam depends on it. Consciousness has two parts: arousal (are they awake?) and awareness (do they know what is going on?). Start with the least invasive stimulus and escalate only as needed — voice at normal volume, then loud voice, then touch, then a central painful stimulus such as a trapezius squeeze or supraorbital pressure. Sternal rub is also a central stimulus, but it is no longer recommended as a routine tool because repeated rubbing bruises the skin. What you must not do is substitute a peripheral stimulus — nail-bed or nail-fold pressure — for a central one when you are scoring the motor response, because a limb can withdraw on a spinal reflex arc with no brain involvement at all.

AVPU is the fast screen: Alert, responds to Voice, responds to Pain, Unresponsive. The Glasgow Coma Scale is the standardized score, from 3 (worst possible) to 15 (fully intact), built from three components. Score the best response in each category, score each side’s best motor response but chart the best overall, and record the components, not just the total: a GCS of 9 made of E2 V2 M5 is a very different patient from E4 V1 M4, and “GCS 9” alone hides that. A GCS of 8 or less generally indicates an inability to protect the airway and prompts consideration of intubation. A drop of 2 or more points in the total, or any drop in the motor score, is an urgent change.

Eye opening (E)Verbal response (V)Best motor response (M)
4 — Spontaneous5 — Oriented6 — Obeys commands
3 — To sound / speech4 — Confused conversation5 — Localizes to pressure
2 — To pressure3 — Inappropriate words4 — Normal flexion / withdraws
1 — None2 — Incomprehensible sounds3 — Abnormal flexion (decorticate)
NT — Not testable (swelling)1 — None · NT if intubated (chart “T”)2 — Extension (decerebrate) · 1 — None

In the infant and preverbal child, the pediatric GCS substitutes developmentally appropriate responses: verbal 5 is coos and babbles, 4 irritable cries, 3 cries to pain, 2 moans to pain, 1 none; motor 6 becomes spontaneous purposeful movement and 5 becomes withdraws to touch. In critical care, sedation depth is scored separately with the RASS, from +4 (combative) through 0 (alert and calm) to −5 (unarousable) — a sedated patient’s low GCS may be pharmacologic, and you must know which.

Orientation, cognition and the delirium screen

“Alert and oriented ×4” means oriented to person, place, time and situation. Ask, do not assume: name, where we are right now, the month and year, and why they came to the hospital. Orientation is lost in a rough order — time first, then place, then situation, then person — so a patient who does not know the year but knows everything else is early, and a patient who does not know their own name is either profoundly impaired or not organically confused at all.

Screen cognition with the Mini-Cog: three unrelated words to remember, a clock-drawing task (“draw a clock face with all the numbers, then set the hands to ten past eleven”), then recall of the three words. Score 0–5 (one point per recalled word, two points for a normal clock); a total of 2 or less is a positive screen for cognitive impairment, while 3, 4 or 5 is a negative screen. The MMSE is scored out of 30 with less than 24 suggesting impairment; the MoCA is scored out of 30 with 26 or above considered normal and is more sensitive to mild impairment. None of these diagnoses dementia — they trigger further evaluation.

Delirium is the finding students miss most, because a quietly hypoactive delirious patient looks like a cooperative one. Use the Confusion Assessment Method (CAM): delirium is present when there is (1) acute onset or fluctuating course AND (2) inattention, PLUS either (3) disorganized thinking OR (4) altered level of consciousness. Test inattention concretely — months of the year backward from December, or digit span. Delirium is acute, fluctuating and usually reversible; dementia is chronic, progressive and stable hour to hour; depression can mimic both with poor effort and “I don’t know” answers. Also separate aphasia (a language disorder — expressive/Broca with effortful, halting, telegraphic speech and preserved comprehension; receptive/Wernicke with fluent but nonsensical speech and impaired comprehension) from dysarthria (a motor articulation problem with intact language). An aphasic patient may be fully oriented and unable to show you.

Pearl. A patient who was oriented ×4 at 0800 and is pulling at lines at 2000 has not “gotten confused.” They have an acute change in mental status, which is a symptom — of hypoxia, hypoglycemia, infection, retention, pain, drug effect or stroke — until you have ruled those out. Check a glucose, a pulse oximetry and a bladder scan before you reach for the word “sundowning.”

Cranial nerves I–XII

Test them in numerical order so you never skip one. On a routine medical-surgical patient the screening version takes two minutes: pupils, extraocular movements, facial symmetry, hearing to conversation, swallow, shoulder shrug and tongue protrusion. A focused exam — new neurologic complaint, suspected stroke, head injury, cranial surgery — tests all twelve.

NerveHow you test itExpectedAbnormal suggests
I OlfactoryEyes closed, one nostril at a time, familiar non-irritating scent (coffee)Identifies scent both sidesAnosmia — frontal lesion, basilar skull fracture, viral
II OpticVisual acuity (Snellen), gross visual fields by confrontation20/20 corrected; full fieldsHemianopsia — stroke, tumor, optic pathway lesion
III, IV, VI Oculomotor, Trochlear, AbducensSix cardinal directions of gaze; pupil size, shape, direct and consensual reaction, accommodationConjugate gaze; pupils 2–5 mm, round, equal, briskly reactive (PERRLA)Unilateral fixed dilated pupil → CN III compression / herniation — emergency; pinpoint <2 mm → opioids or pontine lesion
V TrigeminalLight touch to forehead, cheek, jaw bilaterally; clench teeth and palpate masseters; corneal reflex (wisp) if indicatedSymmetric sensation; strong equal massetersUnilateral numbness, weak jaw — brainstem lesion, trigeminal neuralgia
VII FacialRaise eyebrows, close eyes tightly, smile, show teeth, puff cheeks; taste anterior two-thirds of tongueSymmetric movementForehead spared → central lesion (stroke); whole side including forehead → peripheral (Bell palsy)
VIII VestibulocochlearWhispered word at 1–2 feet; Weber and Rinne with a 512 Hz tuning fork; balanceHears whisper both sides; Weber midline; air conduction longer than bone (AC > BC)Conductive vs sensorineural loss; vertigo, nystagmus
IX, X Glossopharyngeal, VagusSay “ahh” and watch the uvula and palate; voice quality; gag reflex if indicatedPalate rises symmetrically, uvula midline, clear voiceUvula deviates away from the weak side; hoarseness, wet voice → aspiration risk
XI Spinal accessoryShrug shoulders against resistance; turn head against resistanceStrong, symmetric 5/5Weak trapezius/sternocleidomastoid — nerve injury, neck surgery
XII HypoglossalProtrude tongue; move side to side; say “light, tight, dynamite”Tongue midline, clear articulationTongue deviates toward the weak side; slurred lingual sounds

Lifespan variation in the neurologic exam

Normal is age-dependent, and calling a normal infant finding pathologic (or missing a retained reflex) is a real error. In the newborn, the posterior fontanel closes by 2–3 months and the anterior fontanel by 12–18 months; a bulging tense fontanel in a quiet upright infant suggests raised intracranial pressure, a sunken one suggests dehydration. Primitive reflexes appear and then must disappear: Moro (startle) fades by 3–6 months, palmar grasp by 3–4 months, rooting by 3–4 months, tonic neck by 4–6 months, plantar grasp by 8–12 months. An upgoing great toe on plantar stimulation (Babinski response) is normal until roughly 12–24 months and abnormal after that. Persistence of a primitive reflex past its window, or asymmetry of any of them, is a referral.

In toddlers and children, bowed legs (genu varum) are expected until about age 2–3 and knock-knees (genu valgum) between about 2 and 7 years, peaking around 3–4 — both usually self-correct. In infancy screen the hips for developmental dysplasia with the Ortolani and Barlow maneuvers up to about 3 months, and after that with asymmetric thigh folds, limited hip abduction and a positive Galeazzi sign (unequal knee heights with hips and knees flexed).

In pregnancy, relaxin and progesterone loosen the pelvic ligaments and the center of gravity shifts forward, producing lumbar lordosis, low back pain, a wide-based waddling gait and increased fall risk; carpal tunnel symptoms are common from fluid retention. In the older adult, expect a loss of about 1 cm of height per decade after age 40, mild kyphosis, reduced muscle mass (sarcopenia), slower reaction time, smaller and more sluggishly reactive pupils, diminished vibratory sense in the feet, and ankle jerks that may be symmetrically diminished or absent — that last finding is common and, when bilateral and isolated, not necessarily pathologic. What is never normal at any age: new asymmetry, new confusion, and a new focal deficit.

Interactive Score a Glasgow Coma Scale in its components, compare it against the last check to catch an urgent change, screen the acutely confused patient with the CAM and Mini-Cog, and turn a cranial nerve finding into a location. Open full ↑
Key point

Assess level of consciousness before anything else, chart the GCS in components rather than a bare total, screen every acute mental status change as delirium until proven otherwise, and interpret every neurologic finding against the patient’s age.

Section 4Motor, Cerebellar, Sensory and Reflex Testing

Dermatome map of the human body showing the sensory distribution of each spinal nerve root from C2 through S5 in front and back views
Reference. A dermatome map lets you convert a patient’s reported area of numbness into a specific spinal nerve root. BruceBlaus, CC BY 3.0 (via Wikimedia Commons)

The motor exam — bulk, tone, strength and drift

Assess motor function in four steps. Bulk: inspect and compare muscle mass bilaterally; measure limb circumference at a fixed landmark if you suspect atrophy. Tone: with the patient relaxed, move each limb passively through range and feel the resistance. Expect mild, even resistance. Flaccidity is absent tone (a lower motor neuron finding); spasticity is velocity-dependent resistance that gives way suddenly (clasp-knife, an upper motor neuron finding); rigidity is resistance throughout the range, either lead-pipe or ratcheting cogwheel rigidity of parkinsonism. Strength: graded 0–5 as in Section 1, tested group by group and compared side to side. Involuntary movement: note tremor at rest (parkinsonian) versus tremor that worsens as the hand approaches a target (intention tremor, cerebellar), plus fasciculations, tics and myoclonus.

The most useful single motor screen on a hospital unit is pronator drift. The patient extends both arms forward, palms up, eyes closed, for 20–30 seconds. Downward drift with pronation of one forearm indicates a corticospinal tract lesion on the opposite side of the brain, and it appears when formal strength testing still measures 5/5. It costs thirty seconds and it catches early strokes. Test the legs in the same spirit: heel slides, or holding both legs elevated off the bed and watching for one to sink.

FeatureUpper motor neuron lesion (brain, spinal cord)Lower motor neuron lesion (anterior horn, root, peripheral nerve)
ToneIncreased — spasticityDecreased — flaccidity
ReflexesHyperactive (3+ to 4+), clonus possibleDiminished or absent (0 to 1+)
Plantar responseUpgoing great toe — positive BabinskiDowngoing (flexor) or absent
Muscle bulkNormal early; disuse atrophy lateMarked atrophy
FasciculationsAbsentPresent
Pattern of weaknessWhole limb or one side of the bodyFollows a specific nerve or root distribution

Cerebellar function, balance and coordination

The cerebellum coordinates; it does not generate power. So cerebellar testing asks for accuracy and rhythm, not force. Rapid alternating movements: the patient pats the thigh alternating palm and back of hand as fast as possible, or taps thumb to each finger in sequence. Slow, clumsy, irregular performance is dysdiadochokinesia. Finger-to-nose: the patient alternates touching their nose and your fingertip while you move it; overshooting the target is dysmetria. Heel-to-shin: supine, the patient runs one heel down the opposite shin from knee to ankle; wavering off the line is a cerebellar finding. Gait and tandem walk: ask for heel-to-toe walking along a straight line.

The Romberg test is frequently misdescribed. The patient stands with feet together and arms at the sides, eyes open for about 20 seconds, then eyes closed for about 20 seconds. It is positive when the patient is steady with eyes open but sways markedly or loses balance with eyes closed — and that pattern means proprioceptive (dorsal column) or vestibular dysfunction, not cerebellar disease. A patient who is unsteady even with the eyes open has a cerebellar problem. Stand close enough to catch the patient every single time; a fall during a Romberg test is a preventable injury and it belongs to you.

The sensory exam

Sensory testing requires an alert, cooperative patient with eyes closed, and it is done by comparing symmetric areas side to side and distal to proximal. Do not tell the patient where you are touching. Screen a routine patient at the shoulders, forearms, hands, thighs, lower legs and feet; test in detail only where symptoms, a dermatome or a known lesion sends you. Ask “does this feel the same on both sides?” rather than “can you feel this?”

Primary sensations: light touch with a cotton wisp; pain with the sharp and dull ends of a broken tongue blade or a disposable neuro pin (never a reused sharp); temperature only if pain sensation is abnormal; vibration with a 128 Hz tuning fork placed on a bony prominence such as the great toe interphalangeal joint or the medial malleolus — ask when the buzzing stops; proprioception by moving the patient’s great toe or finger up or down while holding it by the sides and asking which way it moved. Vibration and proprioception travel in the dorsal columns and are the first to go in diabetic peripheral neuropathy and vitamin B12 deficiency, which is why an annual monofilament and vibration check is standard diabetic foot care.

Cortical (discriminative) sensations are tested only when primary sensation is intact, because they require an intact parietal cortex to interpret normal input: stereognosis (identify a coin or key placed in the hand), graphesthesia (identify a number traced on the palm), two-point discrimination, and extinction (touch both sides simultaneously; failing to report one side despite intact single-side sensation suggests a contralateral parietal lesion and often accompanies neglect after a right-hemisphere stroke). Translate the patient’s numb area into a root using the dermatome map: C6 thumb, C7 middle finger, C8 little finger, T4 nipple line, T10 umbilicus, L4 medial malleolus, L5 dorsum of the foot and great toe, S1 lateral foot and heel, S2–S4 perianal saddle.

Pearl. Saddle anesthesia — numbness across the S2–S4 dermatomes where you would sit on a saddle — is never a benign finding. Combined with new urinary retention and bilateral leg weakness, it is cauda equina syndrome, a surgical emergency measured in hours.

Reflexes — the percussion half of the neuro exam

Deep tendon reflexes are elicited by striking a tendon with the flat or pointed end of a reflex hammer, using a loose wrist and letting the hammer fall by its own weight. The limb must be relaxed and the tendon slightly stretched; position, not force, is what makes a reflex appear. If a reflex seems absent, use reinforcement before you chart a zero: have the patient clench the teeth or interlock the fingers and pull apart (the Jendrassik maneuver) at the instant you strike. Grade each reflex, compare bilaterally, and remember that asymmetry is more meaningful than the absolute grade.

GradeResponseInterpretation
0No responseAbsent — lower motor neuron lesion, peripheral neuropathy, spinal shock
1+Diminished, present only with reinforcementHypoactive — may be normal in some older adults if symmetric
2+Brisk, expected responseNormal
3+Brisker than average, no clonusMay be normal in an anxious patient; upper motor neuron lesion if asymmetric
4+Hyperactive with sustained clonusAlways abnormal — upper motor neuron lesion; in pregnancy, think preeclampsia
ReflexWhere you strikeNerve rootsExpected response
BicepsYour thumb over the biceps tendon in the antecubital fossaC5–C6Elbow flexion
BrachioradialisRadius, about 1–2 inches above the wristC5–C6Forearm supination and elbow flexion
TricepsTriceps tendon just above the olecranon, arm hangingC6–C7 (mainly C7)Elbow extension
PatellarPatellar tendon below the patella, knee flexed and danglingL2–L4 (mainly L4)Knee extension
AchillesAchilles tendon with the foot slightly dorsiflexedS1–S2 (mainly S1)Plantar flexion
Plantar (superficial)Stroke the lateral sole from heel to ball, then across the metatarsal headsL5–S1Toe flexion in adults; upgoing toe with fanning is abnormal after about 2 years of age

Test for clonus whenever reflexes are 3+ or 4+: support the knee in slight flexion and briskly dorsiflex the ankle, holding the stretch. Rhythmic beats of plantar flexion follow; a few unsustained beats can be normal, but sustained clonus is an upper motor neuron finding and, in a pregnant patient with hypertension and headache, a warning of impending eclampsia. Chart reflexes concretely: “DTRs 2+ and symmetric at biceps, triceps, patellar and Achilles. Plantar reflexes downgoing bilaterally. No clonus.”

Interactive Sort eleven motor findings into upper or lower motor neuron patterns, grade a deep tendon reflex 0 to 4+ with its nerve roots and clonus rules, and click a body map to convert a numb area into a spinal root โ€” including the saddle that is a surgical emergency. Open full ↑
Key point

Motor, cerebellar, sensory and reflex testing sort a deficit into upper versus lower motor neuron, and the pattern — spastic with 4+ reflexes and an upgoing toe, versus flaccid with absent reflexes and fasciculations — tells you where the lesion lives.

Section 5Red Flags, Serial Checks and Escalation

Note. BE-FAST adds balance and vision to the classic FAST mnemonic and catches posterior-circulation strokes that FAST alone misses.

Musculoskeletal emergencies

Acute compartment syndrome is the one every orthopedic patient is at risk for and the one students must be able to recognize without being told. Pressure rises inside a closed fascial compartment — from fracture bleeding, crush injury, a tight cast or a circumferential dressing — until it exceeds capillary perfusion pressure and the muscle and nerve inside begin to die. Irreversible damage can occur within 4 to 8 hours. The classic six P’s are pain, paresthesia, pallor, pulselessness, paralysis and poikilothermia, but they are not equally useful: the earliest and most reliable signs are pain out of proportion to the injury, pain that increases with passive stretch of the digits, and paresthesia. Pallor, pulselessness and paralysis are late — a palpable pulse does not rule out compartment syndrome. Nursing actions: stop, do not elevate the limb above heart level (that reduces arterial inflow — keep it at heart level), do not apply ice, loosen or ask for the cast to be bivalved or the dressing split, hold nothing back on reporting, and escalate immediately. Definitive care is fasciotomy; diagnostic compartment pressure above 30 mmHg or a delta pressure under 30 mmHg is the surgical threshold.

Other musculoskeletal red flags: fat embolism syndrome, typically 24–72 hours after a long bone or pelvic fracture, presenting as hypoxemia with new dyspnea, acute confusion, and a petechial rash across the chest, axillae and conjunctivae; cauda equina syndrome — saddle anesthesia, new urinary retention or incontinence, bilateral leg weakness and bowel dysfunction — a surgical emergency; suspected deep vein thrombosis with new unilateral calf swelling, warmth and pain (do not rely on Homans sign, which is neither sensitive nor specific); and a hip fracture presenting as a shortened, externally rotated leg in a patient who cannot bear weight after a fall.

Neurologic emergencies

For suspected stroke, use BE-FAST: Balance loss, Eyes (sudden vision change), Face droop, Arm drift, Speech difficulty, Time — establish and document the last known well time, which is the time the patient was last seen at neurologic baseline, not the time symptoms were discovered. Activate the stroke alert, keep the patient NPO until a swallow screen is passed, obtain a point-of-care glucose (hypoglycemia is the great stroke mimic), and get the patient to non-contrast CT. Standard targets are door-to-CT within 25 minutes and door-to-needle for thrombolysis within 60 minutes; IV thrombolysis is generally considered within 4.5 hours of last known well, and mechanical thrombectomy in selected patients up to 24 hours. Deficit severity is scored with the NIHSS, 0 to 42.

Rising intracranial pressure announces itself in level of consciousness first — restlessness, then confusion, then a falling GCS — followed by headache, vomiting, and pupillary changes (a sluggish then fixed, dilated pupil on the side of the lesion from third nerve compression). Cushing triad — rising systolic blood pressure with a widening pulse pressure, bradycardia, and irregular or slowed respirations — is a late sign of impending herniation, not an early warning. Normal pulse pressure is 30–40 mmHg; a pressure of 180/60 in a head-injured patient with a heart rate of 48 is an emergency call, not a documentation entry. Decorticate posturing (flexion toward the core) is bad; decerebrate posturing (extension) is worse; progression from one to the other is deterioration.

Autonomic dysreflexia occurs in patients with spinal cord injury at or above T6: a noxious stimulus below the level of injury triggers sudden severe hypertension (systolic often 20–40 mmHg above the patient’s baseline, sometimes far higher), a pounding headache, bradycardia, flushing and sweating above the lesion with pallor and cool skin below, nasal congestion and anxiety. Act in this order: sit the patient upright and lower the legs, loosen constrictive clothing and abdominal binders, then find and remove the trigger — a distended bladder or kinked catheter is the cause in the large majority, then fecal impaction, then skin pressure — while checking blood pressure every 2–5 minutes and calling for help. Untreated it causes stroke, seizure and death.

ParameterAdult expectedAcross the lifespan
Heart rate60–100 beats/minNewborn 100–180; infant 100–160; toddler 90–140; preschool 80–120; school-age 75–118; adolescent 60–100
Respiratory rate12–20 breaths/minNewborn 30–60; infant 30–53; toddler 22–37; preschool 20–28; school-age 18–25; adolescent 12–20
Blood pressureLess than 120/80 mmHg; pulse pressure 30–40Pediatric BP is percentile-based by age, sex and height; older adults commonly have wider pulse pressures from arterial stiffening
Temperature36.5–37.5 °C (97.7–99.5 °F) oralOlder adults run lower and may not mount a fever with infection; infants lose heat quickly
SpO295–100% on room airChronic lung disease targets are often 88–92% per order
Pupils2–5 mm, equal, round, briskly reactiveSmaller and more sluggish in older adults; up to 1 mm anisocoria may be physiologic if longstanding and reactive
GCS15Pediatric GCS uses age-appropriate verbal and motor responses
Pearl. Compare every neuro check to the previous neuro check, not to a textbook. A GCS of 13 that has been 13 all week is a baseline. A GCS of 13 in a patient who was 15 two hours ago is an emergency wearing the same number.

Serial checks, charting and SBAR

Neurologic checks are ordered at a frequency (every 15 minutes, hourly, every 4 hours) because trend is the finding. Perform them the same way every time, at the same depth, using the same stimulus — otherwise the “change” you detect is your technique. A complete neuro check includes level of consciousness and orientation, GCS components, pupils (size in millimeters and reactivity, both eyes), motor strength in all four extremities, sensation, speech quality and vital signs.

Vague entryPrecise entry a nurse can act on
“Neuro checks WNL. Pupils equal.”“Alert and oriented ×4. GCS 15 (E4 V5 M6). Pupils 3 mm bilaterally, round, brisk to light, consensual intact. Strength 5/5 all four extremities, no pronator drift. Sensation intact to light touch all extremities. Speech clear, no aphasia or dysarthria. BP 128/74, HR 76, RR 16.”
“Patient more sleepy this shift, will monitor.”“1400: GCS 11 (E3 V3 M5), down from 15 at 1200. Right pupil 5 mm sluggish, left 3 mm brisk. Left arm drift with 3/5 grip, right 5/5. BP 176/62 (pulse pressure 114, was 128/78), HR 52, RR 10 and irregular. Rapid response activated 1403, provider notified 1404.”
“Complains of cast pain, medicated.”“2210: Right forearm pain 9/10, unrelieved 45 min after oxycodone 5 mg, described as deep and burning, markedly worse with passive finger extension. Fingers cool and dusky compared to left, cap refill 4 sec (left 2 sec), numbness in index and middle fingers, radial pulse palpable. Cast intact and snug. Limb kept at heart level, not elevated. Provider paged 2212.”

When you escalate, use SBAR so the person on the other end of the phone gets what they need in fifteen seconds. Compartment syndrome example: S — “This is Jordan Ellis, RN, on 4 West. I’m calling about Mr. Alvarez in 412, post-op day one right radius ORIF with a long-arm cast. I am concerned about acute compartment syndrome.” B — “He fell yesterday, had a closed distal radius fracture repaired at 1500, has had a well-controlled pain score of 3 to 4 all evening.” A — “Since 2130 his pain is 9 out of 10, unrelieved by oxycodone, and severely worse with passive extension of his fingers. He has new numbness in the index and middle fingers, cap refill 4 seconds on the right versus 2 on the left, fingers cool and dusky. Radial pulse is still present. I have kept the arm at heart level and removed nothing.” R — “I need you to evaluate him now for compartment syndrome and give me an order to bivalve or remove the cast. Can you come to the bedside, or should I activate the rapid response team?”

Stroke example: S — “Room 216, Mrs. Chen, new left-sided weakness and slurred speech, I am calling a stroke alert.” B — “72-year-old admitted for pneumonia, atrial fibrillation, on apixaban, baseline oriented ×4 and independent.” A — “Last known well 1830 when I gave her medications. At 1905 she has left facial droop with forehead sparing, left arm drift with 2/5 grip, left leg 4/5, dysarthric speech, BE-FAST positive. Glucose 112. BP 168/88, HR 96 irregular. NPO, head of bed 30 degrees, IV patent.” R — “I need stat CT and the stroke team at the bedside now; please confirm the code stroke activation.”

Delegation and scope

The Five Rights of Delegation are right task, right circumstance, right person, right direction and communication, right supervision and evaluation. The rule that never bends: the RN may delegate a task, never the nursing process. Assessment, interpretation, teaching, evaluation, clinical judgment and any patient who is unstable or changing stay with the RN. A UAP may collect data and report it; only the RN may decide what it means.

May be delegated to UAPStays with the RN (or LPN/LVN per state scope)
Ambulating a stable patient with a specified device and level of assistanceThe initial and any ongoing musculoskeletal or neurologic assessment, including the first ambulation after a change in status
Performing taught passive or active-assist range of motion on a stable patientDetermining that range of motion is safe, and interpreting new pain or resistance during it
Applying an ordered immobilizer, splint or compression device that is already fittedNeurovascular checks distal to a fracture, cast or surgical site — and the decision that a limb is compromised
Repositioning, transferring with a lift, applying a gait belt, sitting with a fall-risk patientFall-risk assessment and the plan of care that follows it; teaching the patient and family
Measuring and reporting vital signs and pulse oximetry on a stable patientNeuro checks and GCS scoring, orthostatic vital signs on a symptomatic patient, and any vital sign on an unstable patient
Reporting exactly what was observed — “he could not lift his left arm during his bath”Deciding what that observation means, escalating it, and documenting the assessment
Pearl. When you delegate, name the report-back trigger out loud: “Walk Mr. Alvarez to the bathroom with the gait belt and the walker, and come get me immediately if he is dizzy, if his knees buckle, or if he says his right hand is more numb.” A UAP who has been told exactly what to report will report it. One who has not, will not.
Interactive Triage eleven real findings into routine, notify the provider or rapid response and see exactly what the miss would have cost, assemble a scored SBAR escalation from a statement bank, then decide what a UAP may do and what never leaves the RN. Open full ↑
Key point

Pain out of proportion under a cast, a new focal deficit, a falling GCS with a widening pulse pressure, saddle anesthesia with retention, and severe hypertension with headache in a T6-or-above spinal cord injury are all escalations now — delivered in SBAR, by the RN, with a specific request attached.

The chapter in one breath

The musculoskeletal and neurologic exams are built the same way: inspect, palpate, percuss and auscultate — the sequence used in every region except the abdomen, where auscultation moves ahead of percussion and palpation so the bowel is heard before it is provoked — then, for joints, add active range of motion, passive range of motion and strength graded 0 to 5, always compared left to right. Range of motion is charted in degrees; effusion, warmth and crepitus are found with your hands; gait and the Timed Up and Go (12 seconds or more equals fall risk) tell you whether any of it works. On the neurologic side, level of consciousness comes first, scored as a GCS documented in its components, followed by orientation, delirium screening with the CAM, cranial nerves I to XII, motor tone and pronator drift, cerebellar coordination, sensation mapped to dermatomes, and reflexes graded 0 to 4+ with 2+ expected. The patterns then sort themselves into upper versus lower motor neuron. Findings only matter if they are trended and charted concretely and, when they are red flags — compartment syndrome, cauda equina, autonomic dysreflexia, stroke, rising intracranial pressure — escalated immediately in SBAR by the RN, who may delegate the task but never the assessment.

Words worth knowing

TermWhat it means
Active range of motionMovement the patient produces themselves; tests joint, muscle, nerve and willingness together.
Passive range of motionMovement you produce for a relaxed patient; isolates the joint itself.
CrepitusPalpable or audible grating within a joint or under the skin during movement.
Bulge signBedside maneuver that reveals a small knee effusion by milking fluid from one side of the patella to the other.
Antalgic gaitA limp in which the patient shortens time spent bearing weight on the painful limb.
Trendelenburg signPelvic drop on the unsupported side during single-leg stance, indicating gluteus medius weakness on the standing side.
Timed Up and Go (TUG)Rise, walk 10 feet, turn, return, sit; 12 seconds or more indicates increased fall risk.
Neurovascular checkSerial assessment distal to an injury or cast — colour, temperature, capillary refill, pulses, sensation, movement, pain and edema.
Compartment syndromeRising pressure inside a closed fascial space causing ischemia; earliest signs are pain out of proportion, pain on passive stretch and paresthesia.
Glasgow Coma Scale (GCS)Standardized score of eye, verbal and motor response, 3 to 15; 8 or less signals inability to protect the airway.
Pronator driftDownward drift and pronation of one outstretched arm with eyes closed — a sensitive sign of a contralateral corticospinal lesion.
DysmetriaOvershooting or undershooting a target on finger-to-nose testing; a cerebellar sign.
Romberg testStanding with feet together, eyes open then closed; increased sway with eyes closed indicates proprioceptive or vestibular dysfunction.
Babinski responseGreat toe dorsiflexion with fanning on plantar stimulation; normal until about 2 years of age, an upper motor neuron sign after that.
ClonusRhythmic involuntary beats of plantar flexion after brisk dorsiflexion; sustained clonus is always abnormal.
DermatomeThe skin region supplied by a single spinal nerve root, used to localize sensory loss.
AphasiaLoss of language — expressive (halting, effortful) or receptive (fluent but meaningless) — distinct from dysarthria, which is slurred articulation.
Cushing triadRising systolic pressure with widening pulse pressure, bradycardia and irregular respirations; a late sign of rising intracranial pressure.
Autonomic dysreflexiaSevere hypertension with headache and flushing above the lesion in spinal cord injury at or above T6, triggered by a noxious stimulus below it.
Cauda equina syndromeSaddle anesthesia, urinary retention or incontinence and bilateral leg weakness from compression of the lumbosacral nerve roots; a surgical emergency.

Check yourself

Why is the assessment sequence reordered in the abdomen, and what is the order everywhere else?
Everywhere else the order is inspection, palpation, percussion, auscultation. In the abdomen it becomes inspection, auscultation, percussion, palpation, because percussing and palpating first stimulates the bowel and changes the bowel sounds you are about to auscultate. Only the abdomen changes; the musculoskeletal exam keeps the standard order and then adds active range of motion, passive range of motion and graded strength.
A patient can move the elbow through full range only when you support the forearm sideways on the bed, but cannot lift it against gravity. What grade is that, and what does it mean?
That is 2/5 — full range of motion with gravity eliminated. Grade 3/5 requires full range against gravity, 4/5 adds some resistance and 5/5 full resistance. A 2/5 finding is significant weakness and, if new, must be reported and compared with the previous documented grade on the same side.
On active range of motion the shoulder abducts only 70 degrees, but you can passively move it to 180 degrees with no resistance. Where is the problem?
Not in the joint. Full passive range proves the joint surfaces, capsule and cartilage allow the movement. Limited active range with full passive range points to muscle, nerve, pain or effort — think rotator cuff tear, axillary nerve or C5 root injury, a central lesion, or simply severe pain limiting effort. Document both values separately.
A 78-year-old completes the Timed Up and Go in 16 seconds. What does that mean and what do you do?
12 seconds or more indicates increased fall risk, so 16 seconds is a positive screen. Communicate it to the team, place the patient on the fall-risk protocol (bed low, call light in reach, non-skid footwear, purposeful rounding), assess gait, footwear, vision and medications, obtain orthostatic vital signs, and arrange physical therapy evaluation. Also ask the three key questions about falls in the past year, unsteadiness and fear of falling.
Two hours after cast application for a tibial fracture, a patient reports pain of 9/10 unrelieved by opioids and screams when you passively extend the toes. The pedal pulse is palpable. What is your interpretation and your action?
Pain out of proportion plus pain on passive stretch is acute compartment syndrome until proven otherwise, and a palpable pulse does not rule it out — pulselessness is a late sign. Keep the limb at heart level (do not elevate it above the heart and do not ice it), notify the provider immediately for cast bivalving or removal and pressure measurement, continue neurovascular checks, keep the patient NPO in case of fasciotomy, and document times. Irreversible damage can occur in 4 to 8 hours.
Your patient’s GCS is 9. Why is that number, by itself, inadequate documentation?
Because the same total can describe very different patients. E2 V2 M5 (localizes to pressure) is far less ominous than E4 V1 M4, and the motor score carries the most prognostic weight. Chart the components — “GCS 9 (E2 V2 M5)” — along with the stimulus you used, so the next nurse can reproduce the exam and detect a real change rather than a difference in technique.
A patient has a facial droop that spares the forehead — they can still wrinkle it. What does forehead sparing tell you?
It points to a central lesion, such as a stroke, because the upper face receives bilateral cortical innervation and is therefore preserved when one hemisphere is affected. Weakness of the entire half of the face including the forehead points to a peripheral facial nerve (CN VII) lesion such as Bell palsy. Combine that with arm drift and speech change and treat it as a stroke alert.
Describe a positive Romberg test and say what system it implicates.
The patient stands with feet together and arms at the sides, steady with eyes open, then sways markedly or loses balance within about 20 seconds of closing the eyes. That pattern implicates proprioception (dorsal columns) or the vestibular system — not the cerebellum. A patient who is unsteady even with eyes open has a cerebellar problem. Stand close enough to catch them throughout.
What deep tendon reflex grade is expected, and what does 4+ with sustained clonus indicate?
2+ is the expected, normal response. 4+ is hyperactive with sustained clonus and is always abnormal, indicating an upper motor neuron lesion. In a pregnant patient with hypertension, headache and visual changes, 4+ reflexes with clonus suggest severe preeclampsia and impending eclampsia — escalate immediately. Before charting a 0, attempt reinforcement with the Jendrassik maneuver.
Rewrite this entry so it is usable: “Pt confused this evening, redirected, will continue to monitor.”
Something like: “2000: Oriented to person only; at 1200 was oriented ×4. Unable to recite months backward past October. Pulling at IV, requires redirection every few minutes. GCS 14 (E4 V4 M6 — eyes open spontaneously, confused conversation, obeys commands). Pupils 3 mm equal and brisk. Strength 5/5 ×4, no drift. Glucose 92, SpO2 96% RA, temp 38.4 °C, bladder scan 620 mL. CAM positive. Provider notified 2008.” Acute change plus objective data plus what you already ruled out.
A patient with a T4 spinal cord injury develops a pounding headache, BP 210/110 (baseline 105/65), flushing above the injury and bradycardia. What is happening and what is your first action?
Autonomic dysreflexia, which occurs at or above T6. First action is positional: sit the patient fully upright and lower the legs to use gravity to reduce blood pressure, then loosen constrictive clothing and binders. Next, hunt the trigger — check for a distended bladder or kinked or blocked catheter first (the most common cause), then fecal impaction, then skin pressure or a tight device. Recheck blood pressure every 2 to 5 minutes and call for help; untreated it can cause stroke or seizure.
Which of these can you delegate to a UAP on a stroke unit: ambulating a stable patient with a walker, performing the four-hour neuro check, teaching the family about swallowing precautions, or reporting that the patient’s grip felt weaker during the bath?
You may delegate ambulating the stable patient with the specified device and level of assistance, and you should welcome the UAP reporting the observation about grip strength — observing and reporting is within their role. You may not delegate the neuro check (assessment) or family teaching (education), and interpreting the weak grip is nursing judgment that stays with you. When you delegate, name the exact findings you want reported back.